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Further analysis showed that this impairment of DNA synthesis is associated with alterations in the T-cell receptor (TCR) signaling, including conformational changes in TCR and LCK, reduced PLC-1 phosphorylation and calcium flux, and increased ERK phosphorylation [36]

Herein, we report that, via a TCF-independent mechanism, genotoxic stress induces the enrichment of -catenin in chromatin, where it forms a complex with ATM phosphorylated-JDP2 and PRMT5

Mechanistic studies in mice indicate that IL-17 contributes to hepatic inflammation by inducing CXCL10-dependent macrophage accumulation and enhancing collagen secretion by HSCs via STAT3-dependent pathways (119122)