cigarette smoke mitochondrial dysfunction small airway epithelial cells Disruption of the Molecular Regulation of Metabolism in and Lung by Smoke: Are Aldehydes the Culprit? Targeting mitochondrial permeability transition pore

Targeting mitochondrial permeability transition pore ameliorates PM2.5 induced mitochondrial dysfunction in airway epithelial cells ScienceDirect Cigarette smoke can reprogram cells in your airways, causing pulmonary disease to hang on after smoking ends Frontiers Mechanisms of airway epithelial injury and abnormal repair in asthma and COPD Cigarette Smoke Induced Respiratory Response: Insights into Cellular Processes and Biomarkers

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