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Glutamate may mediate the breakdown of BBB in MS and Experimental Autoimmune Encephalomyelitis (EAE) through the action of ONOO, and Studies on EAE suggest that pharmacological inhibition of specific glutamate receptors can suppress neurological symptoms and prevent BBB collapse (Scott et al., 2007)

These findings provide a theoretical foundation for developing drugs targeting apoptosis-related pathways, potentially enhancing treatment precision and efficacy

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The expression levels of Bcl-2, Cyclin-D 1 , and caspase-3 cleavage were evaluated as well