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Moreover, as reported in a previous study 11,12 , ferroptotic cell death is capable of releasing its intracellular components as pro-inflammatory damage-associated molecular patterns (DAMPs), and the releasing and accumulation of DAMPs can trigger tissue inflammation, resulting in the development of various inflammatory diseases (e.g., PE 12 )

Interestingly, one study has highlighted the therapeutic potential of TGFb signaling to rescue midbrain dopaminergic neurons against apoptosis [27]

Maes et al., 2011), and reduced activity of the antioxidant glutathione (GSH) in postmortem brain samples (Gawryluk et al., 2011)

(69) engineered CuP/Er, a nanoparticle that co-delivers Cu 2+ and erastin